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Continue reading LG Spectrum hands-on (video)
LG Spectrum hands-on (video) originally appeared on Engadget on Tue, 10 Jan 2012 18:10:00 EDT. Please see our terms for use of feeds.
Permalink | | Email this | CommentsSource: http://www.engadget.com/2012/01/10/lg-spectrum-hands-on-video/
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ScienceDaily (Jan. 9, 2012) ? Dozens of giant tortoises of a species believed extinct for 150 years may still be living at a remote location in the Gal?pagos Islands, a genetic analysis conducted by Yale University researchers reveals.
The analysis, published Jan. 9 in the journal Current Biology, suggests that direct descendants of at least 38 purebred individuals of Chelonoidis elephantopus live on the volcanic slopes of the northern shore of Isabela Island -- 200 miles from their ancestral home of Floreana Island, where they disappeared after being hunted by whalers.
"This is not just an academic exercise," said Gisella Caccone, senior research scientist in the Department of Ecology and Evolutionary Biology and senior author of the paper. "If we can find these individuals, we can restore them to their island of origin. This is important as these animals are keystone species playing a crucial role in maintaining the ecological integrity of the island communities."
On his historic voyage to the Galapagos in 1835, Charles Darwin observed that the shells of tortoises living on different islands of the chain had different shapes -- one of the observations that inspired his theory of natural selection. For instance, the shells of C. elephantopus on Floreana were saddle-shaped while tortoises on other islands had domed-shaped shells.
On Floreana, however, the tortoises disappeared because of hunting by whalers and workers at a heating oil factory that had been established on the island.
A team of Yale researchers visiting Volcano Wolf on the northern tip of Isabela Island in 2008 took blood samples from more than 1600 tortoises and compared them to a genetic database of living and extinct tortoise species. An analysis detected the genetic signatures of C. elephantopus in 84 Volcano Wolf tortoises, meaning one of their parents was a purebred member of the missing species. In 30 cases breeding had taken place within the last 15 years. Since the lifespan of tortoises can exceed 100 years, there is a high probability that many purebreds are still alive, note the researchers.
"To our knowledge, this is the first report of the rediscovery of a species by way of tracking the genetic footprints left in the genomes of its hybrid offspring," said former Yale postdoctoral researcher Ryan Garrick, now assistant professor at the University of Mississipi and first author of the paper.
Intensive breeding of hybrids might allow scientists to resuscitate the C. elephantopus species even if sufficient numbers of purebred tortoises can not be found, Garrick said.
The team doubts that the tortoises reached Volcano Wolf on their own and theorized the tortoises were transported as food from Floreana, but were either thrown overboard by whalers or left on shore of Isabela.
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Source: http://www.sciencedaily.com/releases/2012/01/120109145723.htm
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Accidente de autob?s en una carretera de Montana deja dos muertos dlvr.it/148sPH Diario de Yucat?n
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— Diario de Yucatán (@DiariodeYucatan) January 8, 2012
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The service will be held at the Victory Tabernacle New Testament Church of God, Port Henderson Road, Bayside, Portmore in St. Catherine.
The officiating minister is the Reverend Bishop Wellesley Blair.
Source: http://go-jamaica.com/news/read_article.php?id=34396
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Contact: Mary Jane Gore
mary.gore@duke.edu
919-660-1309
Duke University Medical Center
DURHAM, N.C. -- Researchers at Duke University Medical Center have developed a new way to identify the genes of harmful microbes, particularly those that have been difficult to study in the laboratory.
This new method uses chemicals to create mutant bacteria, followed by genomic sequencing to identify all mutations. By looking for common genes that were mutated in Chlamydia sharing a particular trait, the investigators were able to rapidly "zero in" on the genes responsible for that trait.
The approach is versatile and inexpensive enough that it could be applied to study a range of microorganisms, said Raphael Valdivia, Ph.D., an associate professor of molecular genetics and microbiology at Duke.
"We were able to learn about genes that allow Chlamydia to flourish in their hosts without the traditional, lengthy process of domesticating the pathogen to accept recombinant DNA," Valdivia said. "Our approach marries classical microbiology techniques with 21st century genome-sequencing technologies. If you encounter a new dangerous microorganism and want to determine what genes are important, I think this represents an effective way to learn all we can, as fast as we can."
One of the goals in studying microbial pathogens that harm humans and animals is to locate and disrupt the genes required for infection, Valdivia said.
The microbe in this study, Chlamydia, is usually sexually transmitted, hides in human cells, and is a type of bacteria that must cause disease to be transmitted from one host to another. Chlamydia is the leading sexually transmitted infection and a risk factor for pelvic inflammatory disease and infertility.
Prior to this work, the function of many Chlamydia genes had to be inferred by their similarity to genes from other bacteria. "By isolating mutants that don't grow well inside cells and identifying the underlying mutations, we can learn a lot about how these genes contribute to disease," Valdivia said. "These are the activities we'd like to block."
"For us, this significantly accelerates the analysis of Chlamydia and importantly, should be applicable to many other microbes that have been difficult manipulate with recombinant DNA approaches," he said. Valdivia suggested that even microbes associated with our normal intestinal flora, which are notoriously difficult to manipulate, are now open to exploration so that we can learn how their genes influence human health, including dietary disorders and inflammatory bowel disease.
The work was published on Jan. 9 in the early edition of the Proceedings of the National Academy of Sciences.
Valdivia also said that the new technique could help to create Chlamydia vaccines that have a combination of mutations that affect the pathogen's virulence. "That way we can cripple some aspects of the bacterium's ability to thrive intact in a host, while still allowing the bacterium to replicate enough to prime the immune system against it."
###
The lead author was Raphael H. Valdivia.
This work was supported by funds from a Chancellor's Science Council Pilot Projects award from Duke University and funds from the NIH.
?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Contact: Mary Jane Gore
mary.gore@duke.edu
919-660-1309
Duke University Medical Center
DURHAM, N.C. -- Researchers at Duke University Medical Center have developed a new way to identify the genes of harmful microbes, particularly those that have been difficult to study in the laboratory.
This new method uses chemicals to create mutant bacteria, followed by genomic sequencing to identify all mutations. By looking for common genes that were mutated in Chlamydia sharing a particular trait, the investigators were able to rapidly "zero in" on the genes responsible for that trait.
The approach is versatile and inexpensive enough that it could be applied to study a range of microorganisms, said Raphael Valdivia, Ph.D., an associate professor of molecular genetics and microbiology at Duke.
"We were able to learn about genes that allow Chlamydia to flourish in their hosts without the traditional, lengthy process of domesticating the pathogen to accept recombinant DNA," Valdivia said. "Our approach marries classical microbiology techniques with 21st century genome-sequencing technologies. If you encounter a new dangerous microorganism and want to determine what genes are important, I think this represents an effective way to learn all we can, as fast as we can."
One of the goals in studying microbial pathogens that harm humans and animals is to locate and disrupt the genes required for infection, Valdivia said.
The microbe in this study, Chlamydia, is usually sexually transmitted, hides in human cells, and is a type of bacteria that must cause disease to be transmitted from one host to another. Chlamydia is the leading sexually transmitted infection and a risk factor for pelvic inflammatory disease and infertility.
Prior to this work, the function of many Chlamydia genes had to be inferred by their similarity to genes from other bacteria. "By isolating mutants that don't grow well inside cells and identifying the underlying mutations, we can learn a lot about how these genes contribute to disease," Valdivia said. "These are the activities we'd like to block."
"For us, this significantly accelerates the analysis of Chlamydia and importantly, should be applicable to many other microbes that have been difficult manipulate with recombinant DNA approaches," he said. Valdivia suggested that even microbes associated with our normal intestinal flora, which are notoriously difficult to manipulate, are now open to exploration so that we can learn how their genes influence human health, including dietary disorders and inflammatory bowel disease.
The work was published on Jan. 9 in the early edition of the Proceedings of the National Academy of Sciences.
Valdivia also said that the new technique could help to create Chlamydia vaccines that have a combination of mutations that affect the pathogen's virulence. "That way we can cripple some aspects of the bacterium's ability to thrive intact in a host, while still allowing the bacterium to replicate enough to prime the immune system against it."
###
The lead author was Raphael H. Valdivia.
This work was supported by funds from a Chancellor's Science Council Pilot Projects award from Duke University and funds from the NIH.
?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Source: http://www.eurekalert.org/pub_releases/2012-01/dumc-nwt010612.php
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Continue reading Mobile Miscellany: week of January 2, 2012
Mobile Miscellany: week of January 2, 2012 originally appeared on Engadget on Sat, 07 Jan 2012 19:00:00 EDT. Please see our terms for use of feeds.
Permalink | | Email this | CommentsSource: http://feeds.engadget.com/~r/weblogsinc/engadget/~3/B3MBrqggl2Q/
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Superstar couple are reportedly calling their first child Ivy Blue.
By Kara Warner
The year's most anticipated new arrival is finally here: Beyoncé Knowles and Jay-Z (nee Shawn Carter) welcomed Ivy Blue Carter, a healthy baby girl, into the world on Saturday (January 7).
The couple's first child was born in New York at St. Luke's Roosevelt Hospital, according to E! News. Beyoncé surprised and delighted fans around the world back in September when she not-so-subtly announced her pregnancy on our very own VMA stage. Rumors have been swirling about the sex of the new bundle of joy since Beyoncé's close friend and former Destiny's Child member Kelly Rowland let it slip that "it" might be a "she." "I have no idea what I'm going to buy Beyoncé at the baby shower because Jay is going to buy that little girl every single thing possible," she told told U.K. news agency Bang Showbiz. "She won't be spoiled, but she will be very well looked-after."
Fans are just as excited for Jay and B's bundle of joy. MTV News spoke to some of them recently, and many said that they expect the newborn to jump right to the top of the celebrity baby power list. "A power baby, obviously," one fan said of the youngster's assumed superstar status, while another added, "It's gonna be a pretty cool baby, that's for sure. Probably gonna run New York City by the time he's 13 or 14." Share your congratulations for Beyoncé and Jay-Z on our Facebook page!
Related ArtistsSource: http://www.mtv.com/news/articles/1676906/beyonce-jay-z-baby-born-ivy-blue.jhtml
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ROME ? No European nation is strong enough to ride out the continent's debt crisis alone, Italy's new premier insisted Saturday, urging fellow EU members to develop a common growth policy.
Premier Mario Monti, leader of the eurozone's third-largest economy, is an economist who was appointed in November with a mandate to pull Italy back from the brink of financial disaster.
"Italy, in order to develop economically and socially, needs Europe, and Europe to be stronger needs Italy," Monti said in a speech in the northern city of Reggio Emilia at a ceremony honoring the Italian flag.
"No European country is so strong that it can go forward alone in facing the great global economies," he added. "Europe needs to put into action common and coordinated growth policies on financial stability."
With Italy making what he called a "decisive contribution" to euro-zone stability, "now it's the time for everyone to do their homework. No one can think they can do less than the others. Europe will overcome the crisis only with the determined and united action of all members," said Monti, a former EU commissioner.
Monti didn't single out any country, but some critics have felt that Germany has been putting its own economic policy ahead of EU-wide interests. Monti will meet with German Chancellor Angela Merkel in Berlin on Wednesday and at a major European summit in Brussels at the end of the month.
EU leaders at that summit will be wrestling with a worsening economic outlook, as more European nations tip over into recession, skepticism keeps rising over many EU countries' bonds and the survival of the euro remains in doubt.
"The eurozone must continue to represent an anchor and a secure reference point in all its geographic extensions," Monti said.
Monti has successfully prodded Italy's often slow-moving parliament into approving quick spending cuts, new and higher taxes and reforms to the long-generous pension system that will see Italians working longer and retiring later.
He singled out two factors in Italy's favor: the fact that many of its families and business "are among the least indebted among industrialized nations." But the premier tried to rally Italians to combat two chronically stubborn problems: corruption and widespread tax evasion by companies and citizens alike.
Foreign investors are frequently discouraged from operating in Italy, where bureaucrats and politicians are often involved in corruption when it comes to securing permits, contracts or funding.
Monti's next priority is spurring growth in Italy, where the economy is stagnant, women have one of the EU's lowest rates of employment and youth joblessness rates run 30 percent nationally and much higher in the underdeveloped south.
But unions have vowed strikes and rallies to protest the government's plan to overhaul labor laws protecting workers, including abolishing a provision that makes it very difficult to fire workers.
Lawmakers, with an eye on 2013 elections, may also be nervous about demanding their voters make financial sacrifices.
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Getty ImagesSunday?s game between the Steelers and Broncos included a bad call on an apparent fumble that allowed the Steelers to force an overtime period from which the Broncos emerged with a memorable win.? Saturday night?s game between the Lions and Saints included a bad call on an apparent fumble that could have allowed the Lions to seize a 21-7 lead late in the first half.
The league admits that two mistakes were made on the play.? One hurt the Lions, and one helped the Lions.
With 5:39 left in the second quarter, Saints quarterback Drew Brees was hit while attempting a pass.? The ball popped forward.? Believing it to be an incomplete pass, an official blew his whistle.? Lions linebacker Justin Durant thereafter recovered the ball and ran toward the end zone.? Then, it was announced that the ruling on the field was that Brees had fumbled ? and that Detroit had recovered.
This prompted outrage from Lions fans, who believed that Detroit had been robbed of a touchdown.? Those Lions fans are right, but it could have been even worse.
?Referee Tony Corrente ruled the play was a fumble and a recovery by Detroit,? the league said in a statement emailed Sunday night to PFT.? ?However, during the play and before Detroit recovered the fumble, another official blew the whistle believing it was an incomplete pass.? Because the ruling on the field was a fumble, and the whistle came before the recovery, the play is dead because of the inadvertent whistle and the Saints should have retained possession of the ball.? New Orleans would then have had the choice to put the ball in play at the spot where possession was lost or to replay the down.? Inadvertent whistles are not reviewable.?
So, basically, the inadvertent whistle took away the Lions? touchdown.? But the rules dictate that the inadvertent whistle also should have given possession back to the Saints, since the ball had not been recovered at the time the inadvertent whistle blew.? (Given the option of taking possession at the spot of the fumble or playing the down over, the Saints would have taken the Mulligan.)
It?s a nonsensical provision, and if the Lions had lost possession of the ball entirely there could have been a ?Fire Millen?-style march upon the league offices.? Given that video is now used to award possession after a whistle is blown in multipe other situations, the league needs to ensure that, if/when last night?s events happen again, the defense will at a minimum retain possession in the event of an inadvertent whistle.
Not because the officiating crew made a mistake in applying the rules, but because that?s what the rules require.
Source: http://profootballtalk.nbcsports.com/2012/01/07/178166/related/
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